EMF Blocking and EMF Shielding

If you reside in an apartment or a house or simply need to ensure that your house is free of EMFs There are plenty ways to reduce exposure. One of the most effective is to restrict the use of electronic devices. It is also possible to use EMF block paint to stop EMF radiation from entering your home. Another way to shield your house against EMF radiations is to put up an RF shielding canopy. It's a kind made of net which contains EMF shielding. It's used to stop EMFs from entering rooms. Another alternative is to get your home equipped with an enclosure that is conductive. They are referred to as Faraday cages.

Numerous studies have demonstrated studies have shown that the nonionizing EMF can cause anti-proliferative effects in HCC cells. The mechanism behind AM RF EMF's anticancer activity in vitro is thought to be based on the downregulation in cancer-related stem cells. This could be the reason for the long-term effects observed in patients with advanced HCC. However, the mechanism behind AM EMF's impact on patients suffering from cancer isn't evident.

Effects on the effects of AM electromagnetic fields (RFEM) on HCC tumor growth in vivo were studied in mice. The tumors were split into 3 groups. First, the group that was unaffected RF EMF. Second group members were exposed RF EMF at frequencies similar to that of humans. Third group members were exposed RF EMF with HCC-specific modulation frequencies. The impact of HCCMF on tumours was assessed against the effect of RCF. The results indicated that tumors treated by HCCMF were significantly shrinking. However, tumours treated with RCF didn't show evidence of shrinkage in the tumour.

The reason for cancer-specific AM RF EMF could be due to the fact that tumor cells require Cav3*2 voltage calcium channels to stimulate growth and down-regulation. AM RF EMF's ability to inhibit proliferation upon HCC cells is caused through CACNA1H the protein which is responsible for the influx of Ca2+ specific to tumours. The results suggest that CACNA1H may have more broader implications in the detection and therapy of the various types of cancer.

 

The tumors of those in the group that were unaffected EMF from radiofrequency, and fed a normal diet for mice. The tumours of HCCMF HCCMF group were treated with Huh7 cells at the time they were between five and seven weeks old. The tumors were then killed after they had a high burden.

The tumors in the three groups also displayed distinct growth curves. The tumors treated with HCCMF had a significant reduction in size of the tumor after 8 weeks. However, the tumors which were treated by RCF showed no signs of shrinkage. The difference was notable. The tumors treated with RCF were able to show necrosis, which is typical when tumors are that are exposed to RCF. The possibility is that the necrosis was due to the lack of oxygen in larger tumors.

In conclusion, the findings suggest an AM-RF EMF is a powerful source of anticancer effects in vitro and in the vivo. Numerous studies have demonstrated the fact that AM RF EMF produces measurable reduction in tumours for HCC patients. There is a possibility that AM RF EMF causes these effects because of CACNA1H which is a protein that participates in the process of tissue-specific Ca2+ influx. Additionally, AM RF EMF may cause a lasting impact on the development of HCC tumors in living tissue.